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Elsevier Regulated Cell Death in Neurodegenerative Disorders - Academic Press

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£139.79 £135.83 £136.69 £137.56 £138.42 £139.29 £140.15 28 July 2026 13 August 2026 30 August 2026 15 September 2026 02 October 2026
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Price range: £136 - £140

Price levels: 2 different prices over 67 days

Description

Regulated Cell Death in Neurodegenerative Disorders is a comprehensive academic resource that examines the mechanisms of RCD (regulated cell death) and its critical role in neurodegenerative diseases. This book explores multiple forms of cell death pathways including apoptosis, necroptosis, ferroptosis, pyroptosis, and autophagy-mediated cell death, each examined within the context of major neurological conditions such as Alzheimer's disease, Parkinson's disease, Multiple sclerosis, Amyotrophic lateral sclerosis, and Huntington's disease. Written by leading experts in the field, each chapter provides specialised insights into the molecular mechanisms of cellular demise and death pathways, alongside current understanding of treatment options and therapeutic approaches for managing these conditions. This reference is essential for neuroscientists, clinical researchers, and healthcare professionals seeking an in-depth understanding of how regulated cell death processes contribute to neurological pathology and disease progression.

Key Features

Regulated Cell Death in Neurodegenerative Disorders covers apoptosis, necroptosis, ferroptosis, pyroptosis, and autophagy-mediated cell death mechanisms in comprehensive detail.

The book examines RCD mechanisms in Alzheimer's, Parkinson's, Multiple sclerosis, Amyotrophic lateral sclerosis, and Huntington's disease with disease-specific analysis.

Written by leading experts who provide unique insights into cellular mechanisms, treatment options, and emerging therapeutic strategies for neurodegenerative conditions.

Academic Press publication offers current research-backed information suitable for neuroscientists, researchers, and clinical practitioners in neurology and internal medicine.

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