£106.59

Humana Mitochondrial Inhibitors and Neurodegenerative Disorders (Contemporary Neuroscience)

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£107 today · all-time low £104 (Jun 2024) · usually the usual

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Price History & Forecast

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Last 608 days • 608 data points (No recent data available)

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£110.97 £103.38 £105.04 £106.69 £108.35 £110.00 £111.66 09 June 2024 07 November 2024 08 April 2025 07 September 2025 06 February 2026

Price Distribution

Price distribution over 608 days • 5 price levels

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Current Price
38 days 265 days 266 days · current 8 days 31 days 0 67 133 200 266 £104 £106 £107 £110 £111 Days at Price

Price Analysis

Most common price: £107 (266 days, 43.8%)

Price range: £104 - £111

Price levels: 5 different prices over 608 days

Description

Mitochondria have long been the Rodney Dangerfield of cellular organelles. Believed to be the remnants of bacterial infection of eukaryotic cells eons ago, the mitochondrion evolved a symbiotic relationship in which it dutifully served as the efficient source of A TP for cell function. The extraordinary dependence of cells on the energy provided by mito chondrial oxidative metabolism of glucose, especially through critical organs such as the heart and brain, is underlined by the fatal consequences of toxins that interfere with the mitochondrial electron transport system. Consistent with their ancestry, the mitochondria have their own DNA that encodes many but not all of their proteins. The mitochon dria and their genes come from the mother via the ovum since sperm do not possess mitochondria. This extranuclear form of inheritance derived exclusively from the female side has proven to be a powerful tool for tracing the evolution by the number of base substitutions in mtDNA. That mitochondrial gene mutations might be a source of human dis ease became evident a decade ago with the characterization of a group of multisystem disorders, typically involving the nervous system, which are transmitted from mother to child. Specific point mutations in mtDNA have been associated with the different syndromes.

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