£180.00

Elsevier Parkinson's Disease and Related Disorders Part I (Volume 83) (Handbook of Clinical Neurology, Volume 83)

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The two volumes on Parkinson's disease in the "Handbook of Clinical Neurology" (Series Editors: Michael J. Aminoff, Department of Neurology, University of California, San Francisco, USA; Francois Boller, Bethesda, USA; Dick F. Swaab, Netherlands Institute for Neuroscience, Amsterdam, the Netherlands) cover a group of disorders that constitute the commonest of neurodegenerative diseases and one that is assuming ever greater importance with the aging of the population in developed countries. The coverage reflects the dramatic advances in understanding of the biochemical background of parkinsonism and the resulting developments in the pharmacological management of the disease.These volumes give a comprehensive account of the subject for both clinical neurologists and those researching in the neurosciences. Part I covers the scientific background, general aspects of Parkinson's disease, clinical aspects and etiology. Part II covers the medical and surgical treatment of Parkinson's disease, complications of therapy and the other parkinsonian syndromes. Review This volume is part of the Handbook of Clinical Neurology series, the world’s most comprehensive source of information in neurology. Now in its third generation, the series has an unparalleled reputation for providing the latest foundational research, diagnosis, and treatment protocols essential for both basic neuroscience research and clinical neurology. From the Back Cover This volume provides a complete guide to advancements made in the understanding of Parkinsons disease and related neurodegenerative disorders. From information on early research conducted in the 1960s, to modern clinical views of these diseases, this handbook will give clinicians and neuroscientists a state-of-the-art reference that presents critical appraisals on recent developments in the study of these disorders. As the world population in developed countries ages, these observations are particularly relevant. Dramatic data on the genetic causes of dominant and recessive forms of hereditary Parkinsons disease are presented, as well as new knowledge on the differences in the intricate circuitry of basal ganglia in healthy individuals and those afflicted with Parkinsons. Clinicians will find this to be a great platform on which to base ongoing investigative approaches and research.

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