£106.12

Springer - Synaptic Plasticity and Alzheimer's Disease Book

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Description

Explore the complex relationship between amyloid-beta protein aggregation and brain function in this specialized scientific volume. Published by Springer, this book examines the biochemical hypothesis that Alzheimer's disease is a progressive cerebral amyloidosis. By focusing on the aggregation of the amyloid b-protein (Ab), the text provides a foundation for understanding the etiologies of the disease. This research collection brings together leading investigators to explore the structure and function of hippocampal synapses. It specifically addresses how early assemblies of Ab may compromise these vital synaptic connections. For professionals in neurology and clinical neurophysiology, this work offers a detailed record of current perspectives on how amyloid-beta affects the brain's ability to maintain synaptic health. This volume is an essential resource for those studying the mechanisms of neurodegeneration and the biological processes behind Alzheimer's disease.

Key Features

Examines the biochemical hypothesis that Alzheimer's disease is a progressive cerebral amyloidosis caused by amyloid b-protein (Ab) aggregation.

Connects research on hippocampal synapse structure and function with studies on how early Ab assemblies impact these connections.

Provides a specialized record of investigations into the etiologies of Alzheimer's disease and neurodegeneration.

Brings together leading investigators focused on the relationship between protein aggregation and synaptic compromise.

Serves as a technical resource for professionals in neurology, internal medicine, and clinical neurophysiology.

Product Specifications

Format
paperback
Domain
Amazon UK
Release Date
30 November 2010
Listed Since
01 October 2010

Barcode

No barcode data available

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